来分cs题目了 档案全文在数位教学平台生理学的临床资料 找不到的可以敲我 我再传给你们 一共十三题 刚好十三人 所以就照学号来吧 刘臻Q1: What vital signs or symptoms do Patient A and Patient B have? 承恩Q2: What neuron in the CNS controls skeletal muscle contraction? Is this kind of neurons’ activities also controlled by interneurons? 博尧Q3. Please draw the contractile structure of the skeletal muscles and draw a neuromuscular junction (NMJ). 铭展Q4. What neurotransmitter is used at the N-M junction? How is the neurotransmitter released and recycled? And how does that release excite skeletal muscle contraction? 柔慧Q5: What is end-plate potential? 明方Q6. Please describe the excitation-contraction coupling events (including the cross-bridge cycle) in the skeletal muscles and how Ca++ ions are involved. Q7: From questions 2 and 6, please list the possible sites along the activation of the motor neuron axon to the contraction of muscles, and describe defects in which step(s) might cause muscle contraction problems? Q8. What is tetanus infection? How would the tetanus infection cause Patient A’s spastic contraction? What is myasthenia gravis (MG)? How would MG cause Patient B’s muscle weakness? Q9: How could decline in muscle function (muscle weakness) contribute to the eye and visual problems and chewing problems of Patient B? Why is neostigmine used as the treatment for Patient B? Q10: Are there any ways to confirm that patient B’s muscle weakness is not due to nerve conduction problem or defects in muscle itself? Q11: What are single fiber electromyography (SFEMG), repeated nerve stimulation (RNS), chest CT scan, tensilon test and serum test? Why are the physiological significances in using these tests for myasthenia gravis diagnosis? Does Patient A need to so those tests? Q12: Have you ever heard of Lambert-Eaton myasthenic syndrome (LEMS)? Is there any test that you can do to differentiate myasthenia gravis from Lambert-Eaton syndrome? Q13: One way to treat MG is to remove the thymus. Why? 以此类推 抱歉 我有点忘了顺序 先预订星期三 三四节讨论吧 地点就在看看吧 抱歉 我刚刚忘了我中午要导生聚 看大家有没有课的 请在下面推文吧 要记得做 ppt 和 word档 谢啦~ --

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