我也是看到没人PO，就鸡婆了 资料请到数位学习平台的生理课那边下载 第一组组员有： 李颖灏　何中庸　赖彦呈　江纯豪　林柏安　刘家翔　潘有法 余贯本　尤冠棠　林紘毅　萧含光　黄圣凯　罗翊中　 讨论时间也订在11/4(三)中午十二点到下午五点 地点暂定在PBL教室第八间 ＜以下转自子豪上一篇文章 XD ＞ Q1: What vital signs or symptoms do Patient A and Patient B have? Q2: What neuron in the CNS controls skeletal muscle contraction? Is this kind of neurons' activities also controlled by interneurons? Q3. Please draw the contractile structure of the skeletal muscles and draw a neuromuscular junction (NMJ). Q4. What neurotransmitter is used at the N-M junction? How is the neurotransmitter released and recycled? And how does that release excite skeletal muscle contraction? Q5: What is end-plate potential? Q6. Please describe the excitation-contraction coupling events (including the cross-bridge cycle) in the skeletal muscles and how Ca++ ions are involved. Q7: From questions 2 and 6, please list the possible sites along the activation of the motor neuron axon to the contraction of muscles, and describe defects in which step(s) might cause muscle contraction problems? Q8. What is tetanus infection? How would the tetanus infection cause Patient A's spastic contraction? What is myasthenia gravis (MG)? How would MG cause Patient B's muscle weakness? Q9: How could decline in muscle function (muscle weakness) contribute to the eye and visual problems and chewing problems of Patient B? Why is neostigmine used as the treatment for Patient B? Q10: Are there any ways to confirm that patient B's muscle weakness is not due to nerve conduction problem or defects in muscle itself? Q11: What are single fiber electromyography (SFEMG), repeated nerve stimulation (RNS), chest CT scan, tensilon test and serum test? Why are the physiological significances in using these tests for myasthenia gravis diagnosis? Does Patient A need to so those tests? Q12: Have you ever heard of Lambert-Eaton myasthenic syndrome (LEMS)? Is there any test that you can do to differentiate myasthenia gravis from Lambert-Eaton syndrome? Q13: One way to treat MG is to remove the thymus. Why? 每个人两题 先挑先赢吧 =ˇ= --

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